Kaiping Yang

Kaiping Yang


PH.D. University of Nottingham
B.Sc. Northwest University
Office: Victoria Research Labs, Room A5-132
p. 519.685.8500 x. 55069 
f. 519.685.8186 
e. kyang@uwo.ca
See Publications by Kaiping Yang on PubMed

Areas of research interests include:

  1. Glucocorticoid actions and metabolism in pregnancy/fetal development
  2. Glucocorticoid actions and metabolism in obesity and its associated metabolic disorders
  3. Molecular basis of intrauterine growth restriction
  4. Fetal origins of visceral obesity
  5. Effects of environmental pollutants/toxins on placental function and fetal development

Both in vivo (animal models) and in vitro (cell cultures and human tissues) as well as classic physiological/biochemical and modern molecular, proteomics and functional genomics approaches/techniques are being utilized.


Yang K, Julan L, Rubio F, Sharma A & Guan H (2006). Cadmium reduces 11B-hydroxysteroid dehydrogenase type 2 activity and expression in human placental trophoblast cells. Am J Physiol Endocrinol Metab 290(1):E135-E142.

Julan L, Guan H, van Beek JP & Yang K (2005). PPAR? suppresses 11B-hydroxysteroid dehydrogenase type 2 gene expression in human placental trophoblast cells. Endocrinology 146, 1482-1490.

Guan H, Arany E, van Beek JP, Chamson-Reg A, Thyssen S, Hill DJ & Yang K (2005). Adipose tissue gene expression profiling reveals distinct molecular pathways that define visceral adiposity in the offspring of maternal protein restricted rats. Am J Physiol Endocrinol Metab 288, E663¨CE673.

Guan H, Dy J, Richardson B & Yang K (2005). Identification of two novel allelic variants ofESX1L in the human placenta: lack of an association with intrauterine growth restriction. Placenta 26, 766-772.

Yang K (1997). Placental 11B-hydroxysteroid dehydrogenase: barrier to maternal glucocorticoids. Reviews of Reproduction 2(3), 129-132